Scottie Health

Scottish Terriers are a generally healthy breed, but with some major and minor issues lurking in the gene pool. The information presented here barely scratches the surface, for indepth reading on these and other topics, check out the links at the bottom of this page.

Diseases & Disorders

Cerabellar Abiotrophy (CA)

Von Willebrands Disease

Craniomandibular Osteopathy

Liver Shunt

Bladder Cancer

Epilepsy

Hypothyroidism

Cushings Syndrome

Juvenile Cataracts

Scottie Cramp


Other Health & Genetic Information

Scottish Terrier Puppy Growth Chart

Coat Color Genetics

CA - Cerebellar Abiotrophy

Cerebellar Abiotrophy (CA),  is a rare, slow-to-progress neurological disease that causes loss of coordination. It has been confused, we
believe, with Scottie Cramp over the years. The primary difference is that Cramp is episodic in nature, while CA is constant, never abates.

The STCA HTF is involved with a study to help find the genetic marker for CA, for more information, visit the CA site, as well as the STCA site.

vWD - Von Willebrands Disease

Every good breeder should be screening for vWD. A simple cheek swap sample collection, and the DNA test done by VetGen, will tell you if the tested dog is Clear, a Carrier, or Affected. Affected dogs should never be bred, and many breeders will also spay or neuter all Carriers. A dog who tests as a Carrier can be carefully bred to a Clear dog, and the resultant offspring will either be Clear or Carriers, with no chance of Affecteds. Breeding to an Affected dog will result in Carriers, Affecteds, or Clear, however if a Carrier is bred to an Affected, there will be no Clear dogs produced. To eliminate vWD EVERY breeder must screen their breeding animals, and use the utmost care if utilizing Carriers in their breeding program. Read the article by Carole Fry Owen, from the STCA's Bagpiper, 1996.

CMO - Craniomandibular Osteopathy

Craniomandibular Osteopathy is an inherited disorder characterized by an abnormal growth of the bone of the lower jaw. CMO usually appears between four and seven months. A puppy with CMO usually pulls away, flinches or screams with pain when his mouth is examined, depending on the severity of the disease. Other early symptoms are lethargy, fever and unwillingness to eat. An acutely affected puppy may be unable to open his mouth but mild cases may be misdiagnosed as teething problems or virus symptoms. An accurate diagnosis of CMO requires X-ray confirmation.

CMO is nearly always treatable. Mild cases respond to aspirin or other non-steroidal anti-inflammatory drugs such as ibuprofen or while acute cases may require the use of steroids such as prednisone or prednisolone. Fortunately, as the dog matures, the abnormal bony growth abates and is often undetectable in the adult dog, even by radiography.

Liver Shunt

To date, liver shunts have been reported in fewer than a dozen dogs but there are several lines that have displayed the problem. Most of what we know about liver shunts comes from research done on Yorkshire Terriers, a breed where this problem is widespread. While we know that the problem is inherited, the mode of inheritance is unknown. Vets at the University of Missouri School of Veterinary Medicine have been working trying to answer this question with a colony of Yorkies with repaired liver shunts but have been unable to get any of the bitches pregnant. The Scottish Terrier Club of America is now working with  a DNA test to detect the carrier status of this problem.

Liver shunt can be difficult for a breeder to recognize but is easily diagnosed with a bile acid test. Affected puppies are normally small and unthrifty.  After eating, they can exhibit bizarre behavior caused by a buildup of ammonia in the bloodstream. Surgery can normally correct the condition, depending on the location of the shunt, but it's not always completely effective and it's very expensive, often running into thousands of dollars. Geneticists recommend that dogs who produce liver shunt should not be bred again and that the siblings of an affected dog should also be spayed/neutered.


 

Bladder Cancer

Scottish Terrers are 18% more likely to develop this cancer than any other breed of canine. At the present time, Purdue University, in conjunction with the Scottish Terrier Club of America Health Trust Fund, is collecting data on both affected animals, as well as unaffected animals. The San Francisco Bay Scottish Terrier Club hosted an indepth seminar earlier this year on Canine Cancer. The speakers presented us with enough data to both educate and motivate. A separate article will be presented on the seminar, look for more information in the coming weeks.

Epilepsy

Seizures may be caused by a number of conditions, including low blood sugar, brain tumor, heat stroke, poison, nutritional deficiency and distemper. Classic or idiopathic (meaning "of unknown cause") epilepsy is characterized by recurrent seizures with no active underlying disease process occurring in the brain. This form of epilepsy is not usually seen until a dog is mature, usually between three and five years of age.

In a typical seizure, the dog will salivate excessively. There is usually dilation of the pupils and stiffening of the limbs. The dog may arch its back and paddle its legs. Frequently, the dog's temperature will spike up three to five degrees. Urination or defecation may accompany or follow the episode. Seizures usually last only a minute or two, but severely affected dogs may have longer and more frequent episodes. Dogs who have infrequent seizures do not require treatment. When treatment is required, phenobarbitol, dilantin and primidone are frequently used.


In recent years, there seems to be an increase of reported seizures in Scottish Terriers. While some of the increase may be due to environmental hazards, inherited epilepsy has definitely made inroads into the Scottie gene pool.

Hypothyroidism

Hypothyroidism is an underproduction of hormones by the thyroid gland. Symptoms of hypothyroidism include:
  Abnormal loss of coat (often bilateral and symmetrical), poor coat condition, fading of coat color
  Chronic skin disorders and infections, skin allergies, dry or scaling skin
  Weight gain
  Infertility
  Fatigue, lethargy
  Intolerance of cold

It is important to determine the exact cause of your dog's hypothyroidism before embarking on a course of treatment. Your veterinarian must run a full thyroid panel and have the blood tested at a laboratory which uses canine thyroid values. Do not be tempted to start thyroid treatment without proper veterinary supervision. The balance of the endocrine system is critical to your dog's health and you can cause an otherwise healthy thyroid gland to atrophy by giving medication improperly.

Cushings Syndrome

Cushing's Syndrome is a collection of symptoms caused by an excess of a hormone called cortisol. There are three main causes of Cushing's Syndrome: a tumor on the pituitary gland; a tumor on the adrenal gland; or veterinarians who over-prescribe corticosteroids to treat itching skin. It is, as yet, unknown whether there is an inherited predisposition to Cushing's Syndrome in Scottish Terriers.

A Scottie should be checked for Cushing's if exihibiting the following symptoms:
  Drinking huge amounts of water and urinating frequently
  Losing coat, thinning coat.
  Darkening of the skin pigment
  Muscles atrophy and pot belly.

If your vet finds the following symptoms, Cushing's Syndrome should be suspected:
  Water intake increases many fold, and frequent urination.
  Elevated SGPT.
  Elevated alkaline phophatase level.
  Ratio of urinary cortisol to urinary creatinine is greater than 24

Cushing's Syndrome is usually treated successfully with a drug called Lysodren. Surgery is rarely recommended and radiation therapy, used in humans, is very expensive and rarely available for dogs.


Juvenile Cataracts

To date, only a few dogs have been affected by juvenile cataracts and apparently only one line. In other breeds, a simple autosomal recessive gene triggers congenital juvenile cataracts. In other words, both  parents must be carriers. 


 Scottie Cramp

Scottie Cramp is the most wide-spread hereditary disorder in the breed and it is also the least serious, from the dog's point of view. Affected dogs are normal at rest and exhibit normal ability to walk until they are stressed. Common stimuli are exercise, hunting, fighting, or courtship. As the dog's level of stress increases, his gait begins to change. The forelegs move out to the side and forward rather than straight forward, called winging. The spine in the lumbar area may arch and the rear legs begin to overflex. If the excitement or exercise continues, the dog begins to exhibit a "goose-stepping" gait. If the dog is running, he may somersault and fall. Severely affected dogs may find their ability to walk or run completely inhibited. As soon as the stimulus abates, the symptoms disappear almost immediately.

The severity of symptoms in affected dogs varies widely as does the type of stimulation necessary to trigger an episode. The Scottie's muscles are not cramping and he is not experiencing pain. He has just temporarily lost the ability to coordinate his movements. In essence, the signals from the brain to the affected muscles is scrambled enroute.

Scottie Cramp is present from birth, but it often takes the eye of an experienced breeder to spot it. Affected dogs soon learn to anticipate the onset of cramping and abruptly stop running or playing. By the time such a puppy is grown, he may never exhibit any signs at all. Similarly, an affected dog with a very laid-back personality is less likely to exhibit symptoms than a more active dog

Scottie Cramp is a life long condition, with no worsening as the dog advances in age. Treatment is seldom necessary but, in severe cases, Vitamin E, diazepam and Prozac have all proven to be effective.



Scottish Terrier Puppy Growth Chart

Coat Color Genetics

Purdue University Bladder Cancer Study

STCA ScottiePhile 

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